Shag Rock (Rapanui) at Sumner Christchurch February 2008 Bob Eregli, from Christchurch Library
A floodtide of extraordinarily heated opinion pieces has recently swept in and swirled around treasured landmarks in the healthcare landscape – much the way Spring Tides once swept around Shag Rock at the entrance to Christchurch Harbor in New Zealand on which Shags (cormorants) perched looking for fish to catch at high tide.
The swirling opinions centered initially on whether SSRIs cause Autism. See
- Unsafe Safety Systems – and the linked media comments
- FDA Panel
- Adam Urato
- Something About Mary
But they have risen further and are now swirling about a Does Tylenol Cause Autism rocky outcrop. See
- Health News Review – the comments following this
- Partnerships in HealthCare
- Epidemiology of Autism
- Pregnancy Acetaminophen or Paracetamol
It may be impossible to frame a response in a way that will work for everyone. A lot depends on what gets asked.
Does Tylenol Cause Autism
This question leads to one set of responses. If by Cause Autism is meant that Tylenol causes all cases of Autism then the answer is clearly no Tylenol is not responsible for Autism across the board.
What we now call Autism is a recent creation as shown by an astonishing escalation in the apparent number of cases since 1980. This new autism has very little to do with the Autism that was first described in the 1930s, which, even though there were islands of high functioning or unusual skills, now looks very much like it involves an element of brain damage.
Milder cases of neurodevelopmental delay have since been thrown into the Autism mix. Also thrown in is a pathologization of normal variation, such that many who may be somewhat introverted and anxious are being diagnosed as Autistic.
The term Autism Spectrum Disorder (ASD), now widely used, pretty well concedes the point that it covers a set of what might be totally different conditions with very different causes.
There are incentives for parents of children and adults on their own behalf to seek out an ASD diagnosis as it offers access to benefits and other supports and can create valuable space for someone.
An explosion in disability claims and payments for mental health conditions from younger folk, however, seems to be bringing welfare systems in many countries close to both economic and political collapse. The political collapse pitches some who figure the situation is completely out of control and so many people cannot be truly disabled in an older sense of that word, against others who feel compelled to resist any attempt to roll-back what are viewed as hard-won human rights.
Can Tylenol Cause Autism?
This is a similar sounding question. It translates into does Tylenol have the capacity to in some cases cause something that might get diagnosed as Autism or ASD.
Before tackling this, we need to note that Tylenol is also implicated in causing Attention Deficit Hyperactivity Disorder (ADHD), which can be viewed as close to the opposite to Autism Spectrum Disorder (ASD).
If some cases of ASD are viewed as states that until recently would have been seen as introversion (a normal and for many admirable variation), some of us until recently called extraverts (not as generally likeable as introverts at least in Britain) are now more likely to be diagnosed as ADHD.
The idea that Tylenol might cause two polar opposite conditions seems wrong. Here cause and effect meets a confusing clinical mess. It is not uncommon now for younger people in particular to be diagnosed as having both ADHD and ASD.
This happens partly because neither of these conditions have clear diagnostic tests or solid markers. Anyone can claim them and some are liable to cancel, ghost or deplatform anyone who might cast doubt on whether it’s valid to diagnose so many people in particular with Adult ADHD.
Tylenol & Neurodevelopmental Delay
Unlike tuberculosis, ulcers or diabetes, which they can have, animals do not get diagnosed with ADHD or ASD. Animals do however show neurodevelopmental delay and we do too and whatever about Tylenol and Autism, we need to pinpoint the causes of this.
If Tylenol Can Cause Autism, it seems almost certain that this autism will feature neurodevelopmental delay.
At present medical clinics do not tease out whether some of the folk getting ASD or ADHD diagnoses also have evidence of neurodevelopmental delay.
The problem with the epidemiology studies flying back and forth is that people bringing problems that might be caused by Tylenol to clinics end up mixed into a soup of ADHD and ASD diagnoses that are not caused by Tylenol.
On one side we have Ahlqvist claiming to have run the biggest study ever in Sweden, but whose methodology looks pretty dodgy. On the other side claims by Baccarelli from Harvard and studies by Liew from Yale, also have problems and are not on their own conclusive.
Anyway, epidemiology studies are not often thought as decisive when it comes to deciding cause and effect. The tobacco companies were, for several decades, able to push back against epidemiology studies that looked compelling by arguing they showed correlations and not causation. There are few things a corporation likes better than Epi studies – they are always able to mount their own Epi which can be guaranteed to cast doubt on claims of a link. As tobacco companies put it – Doubt is our Product.
One of the authors on the Ahlqvist study, Brian Lee, has a remarkable track record of involvement in multiple studies of possible medications taken in utero triggering problems, all of which conclude there is no link or a far less serious risk than we once thought. See Brian Lee Orcid.
The case for Tylenol and a capacity to cause Autism does not lie with Epi studies.
Tylenol causes neurodevelopmental delay in the offspring of pretty well all the pregnant animal species tested. It would be unusual if we were spared.
Tylenol has effects on the epigenomic expression of autism susceptibility genes. There are a range of physical factors that have been implicated in triggering autism from nicotine to thimerosal (mercury in vaccines). None have as wide ranging an effect on autism susceptibility genes as Valproic Acid (valproate, Depakote, Epilim) which is widely accepted, even by Viktor Ahlqvist and colleagues, as causing an autism spectrum disorder – called valproate spectrum disorder.
None that is except Tylenol – see slide.
Valproate is an anticonvulsant, and almost all anticonvulsants seem to come with some risk of leading to autism spectrum conditions. It is not widely known but Tylenol has anticonvulsant properties.
Tylenol also has a toxic metabolite – NAPQI – which has been, and perhaps still is the commonest trigger of liver failure needing liver transplants. NAPQI is not produced normally when we take Tylenol. It takes other stressors to trigger NAPQI production, such as alcohol use. While pregnancy is a normal state, it is also physiologically challenging and we do not know enough to be confident nothing can go wrong. Failing to take the problem seriously may prevent us finding some simple remedies to ensure NAPQI is not produced.
Quite apart from the toxic effects for which we depend on expert input, ideally from both sides of the debate, there is another important element that can be understood without a background in biology.
Sensory input from the early nervous system is the main driver of brain development. Sensory input provides the girders or stanchions on which the developing brain is hung. Drugs acting on the serotonin system mute sensory input, often dramatically so. While this can be useful later in life when we are stressed, it is something to take into account as Tylenol, along with SSRIs, works on the serotonin system and those who find it helpful report a sensory muting effect.
Given that some forms of what later gets called autism spectrum disorder in animals and us are linked to a delay or abnormality in brain development, this clear pathway to problems suggests that it may not be irrational to be cautious until we know more.
Who Calls the Shots?
Whose place is it to make the decision to be cautious or not?
Among the most important factors to take into account is that Tylenol offers no beneficial effect to roughly 50% of us. Many of us take ibuprofen like compounds which work obviously and quickly for us in contrast to Tylenol which seems to do nothing for us. At present, every pregnant woman is advised to take Tylenol – even though she may know it does nothing for her.
Is it responsible to withhold information about possible risks from the 50% of women unlikely to get a benefit? The calculus a woman has to make is quite different if she knows she responds well to Tylenol.
Airing these matters is not about guilt-tripping women. Women who are or who are thinking about getting pregnant do more research than any other group and are likely better than almost any other group at weighing the benefits and risks.
Can withholding information that gets in the way of them doing this be justified? Is there really someone who knows better than they do?.
The concerns about Tylenol extend to children up to the age of 2. Our Epigenome remains unstable/open until 2 years of age, after which it become less pervious to outside influences.
Finally, the onus to warn lies with companies not FDA or academics. The onus to warn does not arise when there is a clear cause and effect. It arises when there are grounds to think there might be a problem.
Rachel Weinstein, an epidemiologist working for Johnson and Johnson, seemed acutely aware of this when in early 2018 she wrote a rather frank email to a colleague – see Rachel to Jesse.
Photo of Rapanui Rock, Christchurch Library, Wikipedia, 21 May 2014
Shag Rock might have been there for Cormorants for several centuries more but Christchurch was hit by an earthquake in 2011 and the Rock was reduced to less than half its previous size.
Peter Selley says
It is often claimed that the prevalence of autism is rising because more children being properly diagnosed.
Of the UK nations it is only Northern Ireland that keeps records of the number of schoolchildren with this diagnosis.
The graphs showing the rising prevalence suggest that that explanation is not the whole story.
https://www.health-ni.gov.uk/sites/default/files/2025-05/asd-children-ni-2025.pdf
I do feel that the lobby that has been pushing the role of children’s vaccines as being the major cause of autism have derailed an open-minded investigation into what is causing this worrying epidemic.
Dr. David Healy says
I think there is some consensus about the fashionable element to it that runs in parallel to the ADHD pandemic. The animal data makes a strong case for a drug linkage – such as the link to valproate – although it took 40 years for that to be accepted. There is a relatively solid link to paracetamol – tylenol also but how big a contribution to the overall prevalence is much harder to tell
D
annie says
The figures from NI point out:
During 2024/25, 14% of school aged children diagnosed with autism were from the most deprived Multiple Deprivation Measure (MDM) decile in Northern Ireland9, while almost 7% of children identified with autism were resided in the least deprived decile of the country10. A statistically significant relationship has been found between the proportion of children identified with autism and MDM Decile. This suggests that the difference is unlikely to have occurred by chance alone.
Do pregnant women from the most deprived areas take more Paracetamol?
Who is diagnosing autism in NI?
Are there more cases of ADHD in NI?
Is NI having a Shag Rock moment?
Secretary Kennedy reposted
HHS.gov @HHSGov9h
.
@DrMakaryFDA on the @US_FDA Announcement that Physicians will be Notified that the use of Acetaminophen (“Tylenol”) During Pregnancy can be Associated with Increased Risk of Autism:
https://x.com/HHSGov/status/1972728882177642728
Secretary Kennedy@SecKennedy6h
To elevate America’s health, restore public trust, and reclaim our reputation for integrity and gold-standard science, @POTUS ’s HHS will challenge even the most sacred public health dogmas through open debate and disciplined scientific scrutiny.
Physicians have been notified, about a ‘sacred public health dogma’; it will be a long-shot to call SSRIs in pregnancy, or the ADHD epidemic, ‘sacred’, but some will swear by it..
chris says
“The case for Tylenol and a capacity to cause Autism does not lie with Epi studies.
Tylenol causes neurodevelopmental delay in the offspring of pretty well all the pregnant animal species tested. It would be unusual if we were spared.”
What does neurodevelopmental delay look like as a result of Tylenol; will it lead to more psych medications and inappropriate psychiatric labels that Allen Frances likes to take responsibility for.
“An explosion in disability claims and payments for mental health condition and payments for mental health conditions from younger folk, however, seems to be bringing welfare systems in many countries close to both economic and political collapse.”
And there is an explosion of very genuine people who have been either killed or seriously harmed in many cases perminately harmed to a dreadful life and early death by psych drug treatments most of which are vehemently denied even by their own relatives in some cases. Anne can attest to what happens when one has the fortitude to try and sue. At the very best they are social outcasts due to the seriously mentally ill labels they can’t remove. Could Tylenol and AD’S be part of perpetuating this?
Harriet Vogt says
You’re so right – ‘the term Autism Spectrum Disorder pretty well concedes the point that it covers a set of what might be totally different conditions with very different causes.’ I think they should bin it – and try harder.
It basically takes a complex stew of human variation and reduces it to its lowest common denominators – for example, the NHS defines autism as:
Problems of communication and empathy, easily overwhelmed by sensory stimuli, slow information processing, upset by the unfamiliar, repetitive thinking or behavioural patterns.
https://www.nhs.uk/conditions/autism/what-is-autism/
Yet, go onto tiktok and the variation is far more instructive:
Young women, who exemplify the fashion end of ‘ASD’. Kelly who attributes her childhood sensitivity to loud noises, preference for plain food and obsession with her mini-nail varnish collection – to autism. Or .mi4.x, why however hard she tries – ‘things go badly’.
https://www.tiktok.com/@beautybykellyx/video/7408132359142264097?lang=en
https://www.tiktok.com/@.mi4.x/video/7512535559466716439?lang=en
On a whole different plane of this diagnostic mashup are children with Level 3, or what they helpfully call ‘Serious Autism’, who are literally disabled and need constant care from some pretty amazing parents:
Nathan’s mom– has a pure calming connection with her beautiful but frustrated, non-verbal son.
https://www.tiktok.com/@mom123x/video/7537323918697221389
And Sydney’s Mom, GretelRoxystar, is unbelievably sanguine about replacing the whole wall that her Level 3 autistic daughter has essentially destroyed with the force of of her stimming.
https://www.tiktok.com/@gretelroxystar/video/7380438322071538990
I think you’ve joined what feel like some fundamental dots in the quest for understanding how both SSRIs and APAP can be implicated in Serious Level 3 Autism – and they don’t seem to be dots anyone else is joining.
Obviously you know, one of the defining characteristics of Serious Autism is sensory dysfunction.
‘Many children with level 3 autism have sensory dysfunction, meaning they are either sensitive or non-sensitive to light, sound, touch, smell, and taste. Bright, crowded, or noisy environments can be overwhelming for children in such cases.’
https://www.songbirdcare.com/articles/level-3-autism
And it’s plausible that this extreme sensory disablement explains other symptoms – like being disconnected from others even to the extent of not recognising their presence ,and repetitive self-stimulating behaviours – stimming.
What frustrating about a lot of the observational research – says she as the most superficial of readers – is that the correlations are often between APAP consumption x trimester x ‘clinically assessed and parent reported neurodevelopmental outcomes’ – which means they’re already stuffed into existing muddled diagnostic frameworks like ASD, rather than carefully related to specific symptoms.
Seems to me – non-scientist – that a good use of some of the RFKJr autism research fund would be to investigate both SSRI and APAP effects on the serotonin system and specific aspects of the sensory system development of poor little rodents.
ADHD is imo far less complex – and largely explained in terms of young for year, too much sugar and the imposition of normative quasi-adult behaviours on kids.
chris says
There might be something in this:
Reduced Glx and GABA Inductions in the Anterior Cingulate Cortex and Caudate Nucleus Are Related to Impaired Control of Attention in Attention-Deficit/Hyperactivity Disorder
https://www.mdpi.com/1422-0067/23/9/4677
annie says
This article in The Observer, spends half the article talking about ‘Special Needs’ children.
How did it go from low employment in autistic people, to Richard Branson and dyslexia.
‘Only three in 10 autistic people of working age are in employment. Yet there are many successful individuals with a neurodivergent condition. The entrepreneur Richard Branson describes dyslexia as his “superpower”.
Peter Kyle, the technology secretary, is so severely dyslexic he has a reading age of eight. He thinks it gives him a different perspective on the world.’
https://observer.co.uk/news/national/article/starmers-next-big-battle-reforming-the-system-for-special-needs-children
Autism diagnosis breakthrough
‘In some parts of England children are facing a seven-year wait for an autism diagnosis, compounding their problems at school and increasing emotional difficulties. Now a groundbreaking project is sending specialist teams into primary schools, cross-checking government data to identify pupils most at risk and delivering extra support within weeks.
The Born in Bradford study has been tracking 13,800 children born at the Bradford Royal Infirmary between 2007 and 2010. Researchers analysed data relating to children who had autism diagnosed by the NHS at age 11 and found many traits were already apparent in information collected routinely by schools in reception.
They could predict with remarkable accuracy which children would later receive an autism diagnosis. Pupils with a low score on the Early Years Foundation Stage Profile – an evaluation carried out on all five-year-olds across England – were 50 times more likely to be diagnosed eventually with autism.
Never trust anyone who says ‘mood music’ ..
Jo Ann Cook says
I agree that Tylenol and SSRI’s are a factor in developmental problems in children but we cannot ignore the “fashionable element” and how governments are again routinely deceiving and manipulating children and parents about “mental health disorders” in children. While our Canadian government promoted the idea of an autism epidemic in a 2007 Senate report and lobbied school boards to root out these children and isolate them to special classes, I was aghast as a social work consultant in a large special education department at what was occurring. This came on the heels of a 2006 report by the same body, the Senate, which claimed that twenty per cent of children and youth were mentally ill and needed treatment. Again, schools were pressured to find these students and send them to doctors.
Dr. June Pimm, a psychologist and an autism expert, who was hired as a coordinator for this new department wrote a Op Ed in a local newspaper in 2014 decrying this so-called epidemic. She claimed that developmental delay did not mean a child had autism. She blamed technology and charlatans. She refuted the 1 in 68 at the time and said it was about 1 in 10,000. Here is her article..
https://ottawacitizen.com/entertainment/books/the-autism-story-from-another-point-of-view
As well, in. 2012 Allen Frances, past chair of the DSM in 1994, claimed that ADHD and Autism were harmful fads. The American Psychiatry Association was said to be reexamining their DSM symptom list and noted they could exclude three quarters of children diagnosed with mild symptoms. No changes were made.
Governments need to fess up to their hidden agenda. In Reclaiming Our Children Peter Breggin describes the beginnings of a US government violence prevention plan which aimed to treat children who had troubling behaviors with psychiatric drugs. This societal program was predicted by Aldous Huxley in Brave New World in which he said science would be used to control non compliant children.
Jo Ann
Harriet Vogt says
In 2025 Allen Frances is STILL saying:
“Should Autism Spectrum Be Split Apart?” Definitely, yes. Why? People w severe #Autism neglected in research/care because ‘Autism Spectrum’ so diluted it includes Elon Musk. Other + changes: 1) Tighten autism criteria 2) Less careless diagnosis mild symptoms’
https://x.com/AllenFrancesMD/status/1973376266243022965
One of the many problems with this mashup ‘diagnosis’ he alludes to – is that research funding is being funneled away from those with what’s known as ‘Serious or Profound Autism’, Level 3, literal life-long disablement:
‘The research that parents of children with profound autism wanted to see was worlds away: “What is causing my child to bang her head against the floor? Why is she having seizures? Why doesn’t she eat? Why doesn’t she sleep? How can I teach her to talk?”
Towards, what is, effectively, easy to conduct research into reasonable adjustments for those I’d dare to describe as having particular cognitive styles and sensory sensibilities, rather than a disabling condition on an incoherent spectrum.
There are various obstacles to a commonsense diagnostic restructure:
1. The industry and/or politically induced muddle headedness of disability researchers such as this chap from Harvard:
‘Dr. Ne’eman said, where would you draw the line? The attempt to split the autism spectrum, he said, was akin to trying to “cleave a meatloaf at the joints.”
2. The understandable emotional resistance and outrage of those who feel validated by a diagnosis that falls somewhere within the chaos.
https://www.nytimes.com/2025/10/01/health/autism-spectrum-neurodiversity-kennedy.html?unlocked_article_code=1.qE8.6asR.guRnrVgTO8Bp&smid=nytcore-android-share
Presumably this is why we’re still living in diagnostic Brave New World 13 years after Allen’s first proclamations about – dangerous fads.
“One believes things because one has been conditioned to believe them.” Aldous Huxley, Brave New World (1931). Technology and science notwithstanding – you sometimes have to wonder how much progress human beings have made. My granny would have seen straight through the non sense.
annie says
SSRIs are enveloping everyone in to a disorder. In 1999, when Seroxat was offered to break the cycle of ‘depression and anxiety’, I swallowed it because pressure was put on me from a local psychiatrist who had just been sold it, I could have been his first, to the doctor and the practise nurse, who thought I would be pretty stupid not to swallow it.
When things took an ugly turn later on, and I was offered Prozac. I was incredulous, No. No. No.
Frightful things happened around 2002, and people died.
The common sense approach by Kennedy, Makary and the team, is to try to eliminate anything that has the possibility of causing autism. Hence, the warning on Tylenol, the panel on SSRIs and pregnancy, to eliminate preservatives and adjuvants, to the possibility of spreading out vaccines in early childhood, and importantly, to study SSRIs and violence.
It might be difficult to monitor results from this, as the ‘spectrum’ of autism seems to be widening the net
Johanna says
Just FYI, here is the FDA’s official draft notice of the new warning they anticipate placing on over-the-counter Tylenol:
https://www.fda.gov/news-events/press-announcements/fda-responds-evidence-possible-association-between-autism-and-acetaminophen-use-during-pregnancy
“The FDA is taking action to make parents and doctors aware of a considerable body of evidence about potential risks associated with acetaminophen,” said FDA Commissioner Marty Makary, M.D., M.P.H. “Even with this body of evidence, the choice still belongs with parents. The precautionary principle may lead many to avoid using acetaminophen during pregnancy, especially since most low-grade fevers don’t require treatment. It remains reasonable, however, for pregnant women to use acetaminophen in certain scenarios.”
“Evidence in recent years has suggested a correlation between acetaminophen use during pregnancy and subsequent diagnosis of conditions like autism and ADHD. Multiple large-scale cohort studies, including the Nurses’ Health Study II and the Boston Birth Cohort, find this association. Some studies have described that the risk may be most pronounced when acetaminophen is taken chronically throughout pregnancy.”
It’s far more cautious and “nuanced” than President Trump’s off-the-cuff bluster on the issue. It has gotten close to zero coverage in the mainstream media, which have focused on Trump’s exhortation for pregnant women to “fight like hell” against taking Tylenol and to “tough it out” if they have fever or pain. As Peter Selley noted above, they have focused on tying this recommendation to the anti-MMR vaccine campaign of RFK Jr.. They have also sought to link it to the draconian anti-abortion measures implemented in Republican-ruled states such as Texas (the implication being that the “Tylenol scare” is simply one more part of the Trump administration’s war on women).
The Trump administration itself has done absolutely nothing to publicize the FDA’s quite moderate statement–apparently they’re more interested in charging up their own hard-core supporters by promising them a “cure for autism” thanks to Trump.
annie says
Actually I don’t think it is a ‘quite moderate statement’. It is a warning, which wasn’t there before.
Without Trump expressing his feelings, who would have bothered to listen. Now the whole strataverse is talking about it. This is Trump’s modus operandi, blast it out, and talk later…
The negative feedback has taken over, some might say it is ‘political’. Kennedy and Makary are not popular in some quarters..
Paracetamol is the talk of the town, and why not, you can’t say nobody doesn’t know about it, and I would suggest that the FDA press release will not harm anyone.
Am I alone in taking Tylenol once in my entire lifetime, arriving in Seattle, after a flight with a screaming infant and a relative who insisted on having his absurd radio station blasting in my ear.
Or never feeling the need for Paracetamol, apart from hospitals foistering it upon me.
I sat in my car over the weekend, to charge up my phone, after a prolonged power cut here in Scotland and when my car radio came on a woman was going on about the nonsense of Paracetamol
and how to add to her credentials she used to work for Bill Gates. This was Woman’s Hour, a UK programme, for women, apparently..
Harriet Vogt says
I think both you and Johanna are right – it’s the difference between Trump’s bullying political purpose and the effect of the Trump ‘Tylenol is BAD’ Show on driving salience for the issue of APAP risks in pregnancy.
Whilst I totally agree that quite likely no one would have listened to the sensible precautionary warning the FDA finally released without the preceding political melodrama – who listens to reason? – we can only speculate about the effects of the campaign overall.
To some extent it’s backfired – at the front end anyway. Trump can be painted as such a blustering buffoon – it enabled Saint Barack Obama – and our own profit-centric Wes Streeting – to dismiss the warnings as those of an unqualified idiot, ditto the MHRA. Health Canada etc.
One can but hope that the clamour of disagreement might lodge in the memories of pregnant mothers and cause a moment’s pause. I guess we need the legal wins to start rolling in to galvanise more serious action.
https://www.wsj.com/health/pharma/kenvue-braces-for-wave-of-new-lawsuits-over-tylenols-potential-link-to-autism-2f29658c
A lot of folk on X were confidently quoting the Ahlqvist study as disproving any risks – because it’s BIG, Bigger, the biggest – 2.48m children, ‘population based’, over 25 years etc.
But even a quick skim by a non-scientist raised instant doubts – only 7.4% children were exposed, why are they talking about ‘prescriptions’, what’s this sibling control business?
Anyway, growing weary of the nonsense, Ann Bauer, posted this piece from James Lyons-Weiler – D will know him and obviously the research inside out- but here’s the link for those like me who don’t – I found it quite clarifying:.
https://popularrationalism.substack.com/p/acetaminophen-and-autism-nbc-news?utm_medium=web&triedRedirect=true
I was particularly interested to read about ‘sensorineural endpoints’ – ‘the girders of brain development’ having stuck in my head as critical:
‘In utero or early-life exposure paradigms have also identified sensory-system liabilities. High-dose prenatal acetaminophen in rodents impaired auditory brainstem responses (Graeca & Kulesza 2024; Hear Res 454:109149; PMID: 37798658; DOI: 10.1016/j.heares.2024.109149), and neonatal exposure reduced striatal BDNF with lasting changes in social and exploratory behavior (Blecharz-Klin 2018; Pharmacol Biochem Behav 168:25–32; PMID: 29530595; DOI: 10.1016/j.pbb.2018.03.004). While dose translation to human pregnancy requires caution, convergent sensory-circuit impacts are notable given clinical ASD phenotypes.’
Harriet Vogt says
Being in the eye of the Tylenol storm, you’ll be aware of this, but for anyone who isn’t:
Texas Attorney General now suing J&J/Kenvue.
https://www.bmj.com/content/391/bmj.r2276
Corporates have covered themselves pretty well re acknowledging SSRI harms on drug labels. But not ASD from pregnant consumption – nor properly PSSD. Could US government take them on too?